BUSCADOR RECOLECTA

OER current as a function of oxides loading, comparison between cyclic voltammetry versus chronoamperometry, impedance spectroscopy measurements, Co K-edge XANES spectra, density of states, Mott–Schottky plot, and BET surface area., Peer reviewed

High resolution X-ray reciprocal space maps, and additional TEM and EFM images and analysis., Peer reviewed

La Colección de Crustáceos Marinos (CRUST-IEOCD) forma parte de las CMR-IEO-CSIC (Colecciones Marinas de Referencia del IEO-CSIC), y surgió a partir de la necesidad de tener que identificar ejemplares de crustáceos procedentes de campañas llevadas a cabo por el Instituto Español de Oceanografía en aguas Africanas. Desde 2005 se comenzaron a recolectar ejemplares, siendo el 2011 el año en que se realizó una exhaustiva revisión taxonómica a partir de la cual se creó la colección oficialmente. Se ponen a disposición de GBIF más de 3100 registros (más de 8500 especímenes). Estos registros suponen el 70% del total de ejemplares depositados actualmente en esta colección. Se trata de ejemplares procedentes, en su mayoría, de campañas de arrastre realizadas por el Instituto Español de Oceanografía (IEO) en aguas africanas de Marruecos, Mauritania, Senegal, Gambia, Guinea-Bissau, Guinea, Guinea Ecuatorial, Namibia y Mozambique, otras campañas realizadas en aguas españolas del Golfo de Cádiz o Mar Cantábrico, o en el Banco de Porcupine (Irlanda), y de campañas perteneciente al proyecto EsMarEs (Estrategias Marinas en España). También aloja ejemplares de la década de los 50 del siglo pasado, donados por el Instituto de Ciencias Marinas de Andalucía (ICMAN-CSIC) y del Departamento de Zoología de la UMA. Además, se pretende tener una base de datos molecular (DNA barcode) de todas las especies depositadas en la CRUST-IEOCD, así como el almacenamiento de todos los especímenes voucher empleados para las codificaciones. En la actualidad se cuenta con 7% individuos voucher codificados, Peer reviewed

Details of samples fabrication method and experimental measurement procedures. Detailed characterization and indexation of all 4 individual DWNTs, based on ED, HRTEM, Rayleigh and Raman spectroscopies. PL spectra of (7,6)@(16,6) DWNT measured at 693 nm (1.79 eV) and 700 nm (1.77 eV) and plotted as a function of Raman shift (with 2G+2D Raman peak indicated in the figure). PL spectra of (14,1)@(15,12) DWNT. Raman spectra of individual SC@M (14,1)@(15,12) DWNT, measured at 776 nm (1.60 eV), 850 nm (1.46 eV) and 457 nm (2.71 eV), showing electronic Raman peaks. Resonant Raman excitation map and resonant Raman excitation profiles of (14,1)@(15,12) DWNT in the G-modes range., Peer reviewed

Document S1. Figs. S1 and S2 and Tables S1 and S2. Document S2. Article plus Supporting Material., Peer reviewed

Additional computational studies for the first 10 orbitals of coronene and corannulene with Pt atoms and a relevant discussion., Peer reviewed

Details on film growth and STEM, together with supplementary images and EELS analysis on [110] cross-sectional thin films, [100]-cross sectional 10 nm thin films, and [001] plane-view specimens., Peer reviewed

[Description of methods used for collection/generation of data] Brown rats were sampled from the sewage system during two periods: from December 2016 to November 2017 when difenacoum and brodifacoum were used, and from August 2021 to July 2022 when bromadiolone was used. Because black rats have just recently been detected in Barcelona, we only studied them during the latter sampling period, with samples obtained from a control action carried out in a green urban area. Exon 3 of the Vkorc1 gene was characterized in both species, while exon 1 was additionally analysed in black rats. Synonymous mutations, not resulting in amino-acid changes, were found in both exons, indicating no evidence of anticoagulant resistance in the rats of Barcelona. [Methods for processing the data] Sequences were analyzed using Geneious v. 2020.0.3 (Kearse et al., 2012)., Agència de Salut Pública de Barcelona., VKORC1_Rattussp_mutations.xlsx, Peer reviewed

Figure S1 Relative gene expression of ALP, BSPII, and RUNX2 genes in hBM-MSCs cultured for 3 days in osteogenic condition and COLII gene for 3 days in chondrogenic condition. *** p < 0.01 as compared to PCL, Figure S2 Staining with Alkaline Phosphatase after 7 days of culture of hBM-MSCs on the two types of scaffolds (PCL and PCL-HAp) in an osteogenic medium. Bars represent 4 mm in (A,C) and 50 µm in (B,D), Figure S3 In vivo subcutaneous implantation of a sterilized scaffold for two weeks. (A,B) Macroscopic analyses showing the good integration and vascularization of the implant, (C,D) Haematoxylin-Eosin (HE) staining, yellow zone: implanted membrane; red zones: blood vessels; blue zones: immune cell aggregates; and white asterisk: fibrocytes, Figure S4 Haematoxylin-Eosin (HE) staining (a–h) and histological staining with Alizarin Red S (i–p) for PCL, PCL-HAp, PCL-HA, and PCL-HAp-HA scaffolds subcutaneously implanted for 4 weeks without cells in ICR mice. S4-1 in (a,e,i,m) for the PCL scaffold S4-2 in (b,f,j,n) for the PCL-HAp scaffold; S4-3 in (c,g,k,o) for the PCL-HA scaffold; and S4-4 in (d,h,l,p) for the PCL-HAp-HA scaffold. Black arrowheads indicate vascularization. Black squares indicate the enlarged areas., Peer reviewed

MAS NMR data and additional calculations and experiments., Peer reviewed