BUSCADOR RECOLECTA

1 table., Peer reviewed

Details of each analyte/sample (ZrH2, C4H2I2S, and LDPE); energy ranges across which the INS spectra were integrated; LOD and LOQ for the elemental H analyte when ZrH2, C4H2I2S, and LDPE are used as the spectral calibrant; and INS spectra of 2,5-diiodothiophene and LDPE, as well as corresponding calibration curves., Peer reviewed

Theoretical background; computational details; estimation of the molecular volume; Rabi-splitting for different electron-photon coupling strengths; energy barrier calculated on HF optimized geometries; energy contributions breakdown; energy barrier dependence on the cavity frequency; Cartesian coordinates of the optimized geometries., Peer reviewed

1 table., Supplementary table with templates and name of primers used for PCR amplifications during the construction of the indicated plasmids are shown., Peer reviewed

Text file containing python code., Peer reviewed

Relaxation maps and further dielectric spectra of the polymers investigated., Peer reviewed

1 table., Peer reviewed

Expression profiling by array, Resources available on the publisher's site: https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE29420, The yeast PMR1 (ATP2C1) gene codes for the eukaryotic prototype of a high affinity P-type ATPase required for Ca2+/Mn2+ transport into the Golgi. Cells lacking PMR1 exhibit multiple genetic interactions with genes involved in DNA recombination and replication, a fact that is not yet understood. We find that deletion of PMR1 causes a delay in DNA replication initiation, progression and G2/M transition and induces the transcriptional up-regulation of genes involved in cell cycle regulation, including CLB5 and SWE1. Interestingly, pmr1∆ clb5∆ double mutants exhibit a dramatic delay in DNA replication and increased DNA breakage, while endoreplication and the formation of multi-nucleated, giant yeast is observed in pmr1∆ swe1∆ cells. Because these phenotypes can be attributed to impeded Mn2+-pump function, we provide a model in which Mn2+ interferes with Mg2+ in the nucleus, and vice versa, Mg2+ interferes with Mn2+ in the Golgi. Consequently, cell cycle progression is challenged by aberrant catalytic activities of enzymes involved in replication and protein glycosylation., Peer reviewed

Supplemental material at [url will be inserted by publisher] contains: diffraction pattern and structural data of SnBi2Te4; core levels measurements and a low energy electron diffraction pattern; a linear fit of the topological surface state., Peer reviewed

1. Computational details 3 2. Convex hull of the FeP4 monolayer 6 3. Calculated exfoliation energy of the FeP4 monolayer 6 4. Stability of the FeP4 monolayer 7 5. Phonon dispersion curves of the FeP4 monolayer at 300 K 8 6. Magnetic configurations of the FeP4 monolayer in a 221 supercell 8 7. Partial density of states (PDOS) onto Fe and P atoms of the FeP4 monolayer and PDOS of different P 3p orbitals in the P4 unit 8 8. Fatband structures of the FeP4 monolayer 9 9. Spin charge density and Curie temperature (TC) of the FeP4 monolayer with incomplete P4 units 9 10. COHP curve of the adjacent P-P pairs in the FeP4 monolayer and schematic diagram of the distribution of 3p orbitals of P atoms in quasi-square P4 units 10 11. Magnetic exchange parameters (J1) of the FeP4 monolayer under biaxial strains 10 12. Magnetic moment variations of the FeP4 monolayer under biaxial strains 10 13. Magnetic transition temperatures of the FeP4 monolayer under biaxial strains 11 14. Structural data about the FeP4 monolayer 11 15. The integrated COHP (ICOHP) for the FeP4 monolayer 11 16. Optimized structural information of the FeP4 monolayer with different magnetic orders 12 17. Total energies of different magnetic configurations of the FeP4 monolayer 12 18. Detailed information of the FeP4 monolayer under different biaxial strains 12 19. Structural information of the predicted FeP4 monolayer 13 20. References 14, Peer reviewed