Resultados totales (Incluyendo duplicados): 45302
Encontrada(s) 4531 página(s)
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331826
Dataset. 2022

VIDEO4_ANALYSIS OF ACTOMYOSIN OSCILLATORY DYNAMICS USING A COARSE-GRAINED MODEL.AVI [DATASET]

  • Hernández del Valle, Miguel
  • Valencia-Expósito, Andrea
  • Gorfinkiel, Nicole
  • Martín-Bermudo, María D.
  • Míguez, David G.
Autonomous oscillatory dynamics are ubiquitous at every level in Biology. At the cellular level, one of the most relevant and well characterized examples of periodic behavior is the cyclic assembly and disassembly of actomyosin networks. In Drosophila, these oscillations induce the robust contraction and expansion of individual cells required for correct dorsal closure, while in the follicular epithelium that surrounds the germline, periodic contractions of the basal actomyosin network are required for proper elongation of the egg chamber. While some studies suggest that actomyosin oscillations are driven by upstream signaling or mechanochemical features, we have recently proposed that they arise as a systems property from the competition between two well characterized features of the actomyosin machinery: 1) cooperative assembly of actin networks mediated by Actin crosslinker proteins and 2) tension-induced disassembly of actin networks mediated by myosin motors. Here, we perform experiments in amnioserosa and in the follicle cells of drosophila and simulations using a coarse-grained model of the actomyosin cortex to characterize the properties of the oscillations and how they depend on different features of the system. We also compare model and experiments to study the dynamics of actomyosin flows and the effect of mechanical coupling between cells in the tissue. In conclusion, our model is a powerful tool to study key features of actomyosin oscillations, from the effect of the individual components to network properties and finally supra-cellular organization of the oscillations at the tissue level., Peer reviewed

Proyecto: //
DOI: http://hdl.handle.net/10261/331826
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331826
HANDLE: http://hdl.handle.net/10261/331826
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331826
PMID: http://hdl.handle.net/10261/331826
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331826
Ver en: http://hdl.handle.net/10261/331826
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331826

Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331827
Dataset. 2022

VIDEO5_ANALYSIS OF ACTOMYOSIN OSCILLATORY DYNAMICS USING A COARSE-GRAINED MODEL.AVI [DATASET]

  • Hernández del Valle, Miguel
  • Valencia-Expósito, Andrea
  • Gorfinkiel, Nicole
  • Martín-Bermudo, María D.
  • Míguez, David G.
Autonomous oscillatory dynamics are ubiquitous at every level in Biology. At the cellular level, one of the most relevant and well characterized examples of periodic behavior is the cyclic assembly and disassembly of actomyosin networks. In Drosophila, these oscillations induce the robust contraction and expansion of individual cells required for correct dorsal closure, while in the follicular epithelium that surrounds the germline, periodic contractions of the basal actomyosin network are required for proper elongation of the egg chamber. While some studies suggest that actomyosin oscillations are driven by upstream signaling or mechanochemical features, we have recently proposed that they arise as a systems property from the competition between two well characterized features of the actomyosin machinery: 1) cooperative assembly of actin networks mediated by Actin crosslinker proteins and 2) tension-induced disassembly of actin networks mediated by myosin motors. Here, we perform experiments in amnioserosa and in the follicle cells of drosophila and simulations using a coarse-grained model of the actomyosin cortex to characterize the properties of the oscillations and how they depend on different features of the system. We also compare model and experiments to study the dynamics of actomyosin flows and the effect of mechanical coupling between cells in the tissue. In conclusion, our model is a powerful tool to study key features of actomyosin oscillations, from the effect of the individual components to network properties and finally supra-cellular organization of the oscillations at the tissue level., Peer reviewed

Proyecto: //
DOI: http://hdl.handle.net/10261/331827
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331827
HANDLE: http://hdl.handle.net/10261/331827
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331827
PMID: http://hdl.handle.net/10261/331827
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331827
Ver en: http://hdl.handle.net/10261/331827
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331827

Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331828
Dataset. 2022

DATASHEET1_CALORIE RESTRICTION RESCUES MITOCHONDRIAL DYSFUNCTION IN ADCK2-DEFICIENT SKELETAL MUSCLE.DOCX

  • Hernández-Camacho, Juan Diego
  • Fernández-Ayala, Daniel J. M.
  • Vicente-García, Cristina
  • Navas-Enamorado, Ignacio
  • López-Lluch, Guillermo
  • Oliva, Clara
  • Artuch, Rafael
  • Garcia-Villoria, Judith
  • Ribes, Antonia
  • Cabo, Rafael de
  • Carvajal, Jaime J.
  • Navas, Plácido
ADCK2 haploinsufficiency-mediated mitochondrial coenzyme Q deficiency in skeletal muscle causes mitochondrial myopathy associated with defects in beta-oxidation of fatty acids, aged-matched metabolic reprogramming, and defective physical performance. Calorie restriction has proven to increase lifespan and delay the onset of chronic diseases associated to aging. To study the possible treatment by food deprivation, heterozygous Adck2 knockout mice were fed under 40% calorie restriction (CR) and the phenotype was followed for 7 months. The overall glucose and fatty acids metabolism in muscle was restored in mutant mice to WT levels after CR. CR modulated the skeletal muscle metabolic profile of mutant mice, partially rescuing the profile of WT animals. The analysis of mitochondria isolated from skeletal muscle demonstrated that CR increased both CoQ levels and oxygen consumption rate (OCR) based on both glucose and fatty acids substrates, along with mitochondrial mass. The elevated aerobic metabolism fits with an increase of type IIa fibers, and a reduction of type IIx in mutant muscles, reaching WT levels. To further explore the effect of CR over muscle stem cells, satellite cells were isolated and induced to differentiate in culture media containing serum from animals in either ad libitum or CR diets for 72 h. Mutant cells showed slower differentiation alongside with decreased oxygen consumption. In vitro differentiation of mutant cells was increased under CR serum reaching levels of WT isolated cells, recovering respiration measured by OCR and partially beta-oxidation of fatty acids. The overall increase of skeletal muscle bioenergetics following CR intervention is paralleled with a physical activity improvement, with some increases in two and four limbs strength tests, and weights strength test. Running wheel activity was also partially improved in mutant mice under CR. These results demonstrate that CR intervention, which has been shown to improve age-associated physical and metabolic decline in WT mice, also recovers the defective aerobic metabolism and differentiation of skeletal muscle in mice caused by ADCK2 haploinsufficiency., Peer reviewed

Proyecto: //
DOI: http://hdl.handle.net/10261/331828
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331828
HANDLE: http://hdl.handle.net/10261/331828
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331828
PMID: http://hdl.handle.net/10261/331828
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331828
Ver en: http://hdl.handle.net/10261/331828
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331828

Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331829
Dataset. 2022

IMAGE1_CALORIE RESTRICTION RESCUES MITOCHONDRIAL DYSFUNCTION IN ADCK2-DEFICIENT SKELETAL MUSCLE.JPEG [DATASET]

  • Hernández-Camacho, Juan Diego
  • Fernández-Ayala, Daniel J. M.
  • Vicente-García, Cristina
  • Navas-Enamorado, Ignacio
  • López-Lluch, Guillermo
  • Oliva, Clara
  • Artuch, Rafael
  • Garcia-Villoria, Judith
  • Ribes, Antonia
  • Cabo, Rafael de
  • Carvajal, Jaime J.
  • Navas, Plácido
ADCK2 haploinsufficiency-mediated mitochondrial coenzyme Q deficiency in skeletal muscle causes mitochondrial myopathy associated with defects in beta-oxidation of fatty acids, aged-matched metabolic reprogramming, and defective physical performance. Calorie restriction has proven to increase lifespan and delay the onset of chronic diseases associated to aging. To study the possible treatment by food deprivation, heterozygous Adck2 knockout mice were fed under 40% calorie restriction (CR) and the phenotype was followed for 7 months. The overall glucose and fatty acids metabolism in muscle was restored in mutant mice to WT levels after CR. CR modulated the skeletal muscle metabolic profile of mutant mice, partially rescuing the profile of WT animals. The analysis of mitochondria isolated from skeletal muscle demonstrated that CR increased both CoQ levels and oxygen consumption rate (OCR) based on both glucose and fatty acids substrates, along with mitochondrial mass. The elevated aerobic metabolism fits with an increase of type IIa fibers, and a reduction of type IIx in mutant muscles, reaching WT levels. To further explore the effect of CR over muscle stem cells, satellite cells were isolated and induced to differentiate in culture media containing serum from animals in either ad libitum or CR diets for 72 h. Mutant cells showed slower differentiation alongside with decreased oxygen consumption. In vitro differentiation of mutant cells was increased under CR serum reaching levels of WT isolated cells, recovering respiration measured by OCR and partially beta-oxidation of fatty acids. The overall increase of skeletal muscle bioenergetics following CR intervention is paralleled with a physical activity improvement, with some increases in two and four limbs strength tests, and weights strength test. Running wheel activity was also partially improved in mutant mice under CR. These results demonstrate that CR intervention, which has been shown to improve age-associated physical and metabolic decline in WT mice, also recovers the defective aerobic metabolism and differentiation of skeletal muscle in mice caused by ADCK2 haploinsufficiency., Peer reviewed

Proyecto: //
DOI: http://hdl.handle.net/10261/331829
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331829
HANDLE: http://hdl.handle.net/10261/331829
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331829
PMID: http://hdl.handle.net/10261/331829
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331829
Ver en: http://hdl.handle.net/10261/331829
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331829

Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331830
Dataset. 2022

IMAGE2_CALORIE RESTRICTION RESCUES MITOCHONDRIAL DYSFUNCTION IN ADCK2-DEFICIENT SKELETAL MUSCLE.JPEG [DATASET]

  • Hernández-Camacho, Juan Diego
  • Fernández-Ayala, Daniel J. M.
  • Vicente-García, Cristina
  • Navas-Enamorado, Ignacio
  • López-Lluch, Guillermo
  • Oliva, Clara
  • Artuch, Rafael
  • Garcia-Villoria, Judith
  • Ribes, Antonia
  • Cabo, Rafael de
  • Carvajal, Jaime J.
  • Navas, Plácido
ADCK2 haploinsufficiency-mediated mitochondrial coenzyme Q deficiency in skeletal muscle causes mitochondrial myopathy associated with defects in beta-oxidation of fatty acids, aged-matched metabolic reprogramming, and defective physical performance. Calorie restriction has proven to increase lifespan and delay the onset of chronic diseases associated to aging. To study the possible treatment by food deprivation, heterozygous Adck2 knockout mice were fed under 40% calorie restriction (CR) and the phenotype was followed for 7 months. The overall glucose and fatty acids metabolism in muscle was restored in mutant mice to WT levels after CR. CR modulated the skeletal muscle metabolic profile of mutant mice, partially rescuing the profile of WT animals. The analysis of mitochondria isolated from skeletal muscle demonstrated that CR increased both CoQ levels and oxygen consumption rate (OCR) based on both glucose and fatty acids substrates, along with mitochondrial mass. The elevated aerobic metabolism fits with an increase of type IIa fibers, and a reduction of type IIx in mutant muscles, reaching WT levels. To further explore the effect of CR over muscle stem cells, satellite cells were isolated and induced to differentiate in culture media containing serum from animals in either ad libitum or CR diets for 72 h. Mutant cells showed slower differentiation alongside with decreased oxygen consumption. In vitro differentiation of mutant cells was increased under CR serum reaching levels of WT isolated cells, recovering respiration measured by OCR and partially beta-oxidation of fatty acids. The overall increase of skeletal muscle bioenergetics following CR intervention is paralleled with a physical activity improvement, with some increases in two and four limbs strength tests, and weights strength test. Running wheel activity was also partially improved in mutant mice under CR. These results demonstrate that CR intervention, which has been shown to improve age-associated physical and metabolic decline in WT mice, also recovers the defective aerobic metabolism and differentiation of skeletal muscle in mice caused by ADCK2 haploinsufficiency., Peer reviewed

Proyecto: //
DOI: http://hdl.handle.net/10261/331830
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331830
HANDLE: http://hdl.handle.net/10261/331830
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331830
PMID: http://hdl.handle.net/10261/331830
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331830
Ver en: http://hdl.handle.net/10261/331830
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331830

Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331831
Dataset. 2022

IMAGE3_CALORIE RESTRICTION RESCUES MITOCHONDRIAL DYSFUNCTION IN ADCK2-DEFICIENT SKELETAL MUSCLE.JPEG [DATASET]

  • Hernández-Camacho, Juan Diego
  • Fernández-Ayala, Daniel J. M.
  • Vicente-García, Cristina
  • Navas-Enamorado, Ignacio
  • López-Lluch, Guillermo
  • Oliva, Clara
  • Artuch, Rafael
  • Garcia-Villoria, Judith
  • Ribes, Antonia
  • Cabo, Rafael de
  • Carvajal, Jaime J.
  • Navas, Plácido
ADCK2 haploinsufficiency-mediated mitochondrial coenzyme Q deficiency in skeletal muscle causes mitochondrial myopathy associated with defects in beta-oxidation of fatty acids, aged-matched metabolic reprogramming, and defective physical performance. Calorie restriction has proven to increase lifespan and delay the onset of chronic diseases associated to aging. To study the possible treatment by food deprivation, heterozygous Adck2 knockout mice were fed under 40% calorie restriction (CR) and the phenotype was followed for 7 months. The overall glucose and fatty acids metabolism in muscle was restored in mutant mice to WT levels after CR. CR modulated the skeletal muscle metabolic profile of mutant mice, partially rescuing the profile of WT animals. The analysis of mitochondria isolated from skeletal muscle demonstrated that CR increased both CoQ levels and oxygen consumption rate (OCR) based on both glucose and fatty acids substrates, along with mitochondrial mass. The elevated aerobic metabolism fits with an increase of type IIa fibers, and a reduction of type IIx in mutant muscles, reaching WT levels. To further explore the effect of CR over muscle stem cells, satellite cells were isolated and induced to differentiate in culture media containing serum from animals in either ad libitum or CR diets for 72 h. Mutant cells showed slower differentiation alongside with decreased oxygen consumption. In vitro differentiation of mutant cells was increased under CR serum reaching levels of WT isolated cells, recovering respiration measured by OCR and partially beta-oxidation of fatty acids. The overall increase of skeletal muscle bioenergetics following CR intervention is paralleled with a physical activity improvement, with some increases in two and four limbs strength tests, and weights strength test. Running wheel activity was also partially improved in mutant mice under CR. These results demonstrate that CR intervention, which has been shown to improve age-associated physical and metabolic decline in WT mice, also recovers the defective aerobic metabolism and differentiation of skeletal muscle in mice caused by ADCK2 haploinsufficiency., Peer reviewed

Proyecto: //
DOI: http://hdl.handle.net/10261/331831
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331831
HANDLE: http://hdl.handle.net/10261/331831
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331831
PMID: http://hdl.handle.net/10261/331831
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331831
Ver en: http://hdl.handle.net/10261/331831
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331831

Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331832
Dataset. 2022

IMAGE4_CALORIE RESTRICTION RESCUES MITOCHONDRIAL DYSFUNCTION IN ADCK2-DEFICIENT SKELETAL MUSCLE.JPEG [DATASET]

  • Hernández-Camacho, Juan Diego
  • Fernández-Ayala, Daniel J. M.
  • Vicente-García, Cristina
  • Navas-Enamorado, Ignacio
  • López-Lluch, Guillermo
  • Oliva, Clara
  • Artuch, Rafael
  • Garcia-Villoria, Judith
  • Ribes, Antonia
  • Cabo, Rafael de
  • Carvajal, Jaime J.
  • Navas, Plácido
ADCK2 haploinsufficiency-mediated mitochondrial coenzyme Q deficiency in skeletal muscle causes mitochondrial myopathy associated with defects in beta-oxidation of fatty acids, aged-matched metabolic reprogramming, and defective physical performance. Calorie restriction has proven to increase lifespan and delay the onset of chronic diseases associated to aging. To study the possible treatment by food deprivation, heterozygous Adck2 knockout mice were fed under 40% calorie restriction (CR) and the phenotype was followed for 7 months. The overall glucose and fatty acids metabolism in muscle was restored in mutant mice to WT levels after CR. CR modulated the skeletal muscle metabolic profile of mutant mice, partially rescuing the profile of WT animals. The analysis of mitochondria isolated from skeletal muscle demonstrated that CR increased both CoQ levels and oxygen consumption rate (OCR) based on both glucose and fatty acids substrates, along with mitochondrial mass. The elevated aerobic metabolism fits with an increase of type IIa fibers, and a reduction of type IIx in mutant muscles, reaching WT levels. To further explore the effect of CR over muscle stem cells, satellite cells were isolated and induced to differentiate in culture media containing serum from animals in either ad libitum or CR diets for 72 h. Mutant cells showed slower differentiation alongside with decreased oxygen consumption. In vitro differentiation of mutant cells was increased under CR serum reaching levels of WT isolated cells, recovering respiration measured by OCR and partially beta-oxidation of fatty acids. The overall increase of skeletal muscle bioenergetics following CR intervention is paralleled with a physical activity improvement, with some increases in two and four limbs strength tests, and weights strength test. Running wheel activity was also partially improved in mutant mice under CR. These results demonstrate that CR intervention, which has been shown to improve age-associated physical and metabolic decline in WT mice, also recovers the defective aerobic metabolism and differentiation of skeletal muscle in mice caused by ADCK2 haploinsufficiency., Peer reviewed

Proyecto: //
DOI: http://hdl.handle.net/10261/331832
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331832
HANDLE: http://hdl.handle.net/10261/331832
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331832
PMID: http://hdl.handle.net/10261/331832
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331832
Ver en: http://hdl.handle.net/10261/331832
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331832

Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331875
Dataset. 2023

EXPERIMENTAL DATA FOR MANUSCRIPT "NOVEL FINDINGS ABOUT THE MODE OF ACTION OF THE ANTIFUNGAL PROTEIN PEAFPA AGAINST SACCHAROMYCES CEREVISIAE"

  • Giner Llorca, Moisés
  • Locascio, Antonella
  • Alonso del Real, Javier
  • Marcos López, José Francisco
  • Manzanares, Paloma
The dataset is made available under the Open Database License. Any rights in individual contents of the database are licensed under the Database Contents License. Please, read the full ODbL 1.0 license text for the exact terms that apply. Users of the dataset are free to: Share: copy, distribute and use the database, either commercially or non-commercially. Create: produce derivative works from the database. Adapt: modify, transform and build upon the database. Under the following conditions: Attribution: You must attribute any public use of the database, or works produced from the database. For any use or redistribution of the database, or works produced from it, you must make clear to others the license of the original database. Share-Alike: If you publicly use any adapted version of this database, or works produced from an adapted database, you must also offer that adapted database under the ODbL., This work was supported by grants RTI2018-101115-B-C21, PID2021-125858OB-100 and the Severo Ochoa Excellence Program CEX2021-001189-S funded by MCIN/AEI/ 10.13039/501100011033 and by “ERDF A way of making Europe”. MGL was recipient of a predoctoral grant FPU19/02066 funded by MCIN/AEI/680 10.13039/501100011033 and by “ESF Investing in your future”., Peer reviewed

DOI: http://hdl.handle.net/10261/331875
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331875
HANDLE: http://hdl.handle.net/10261/331875
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331875
PMID: http://hdl.handle.net/10261/331875
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331875
Ver en: http://hdl.handle.net/10261/331875
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331875

Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331918
Dataset. 2022

METAL- AND ALLOY-BASED CORE–SHELL PARTICLES IN NITRATE SENARY SALT WITH LOW THERMAL HYSTERESIS FOR SOLAR THERMAL ENERGY STORAGE [DATASET]

  • Chen, Kai
  • Chung, Feng-Ju
  • Lin, Yen-Ling
  • Lee, Yen-Ta
  • Rodríguez-Laguna, María del Rocío
  • Manikandan; Arumugam
  • Lu, Ming-Chang
  • Chueh, Yu-Lun
3 pages. -- Figure S1 Particle size distribution of (a) Sn, (b) Sn/a-C, and (c) SnBi/a-C MPs. -- Figure S2 (a) A SEM image of core and shell layer of SnBi/a-C MPs. (b) EDX line scan mapping images of Sn, Bi and amorphous carbon along the yellow arrow., In this work, the microencapsulated phase change materials, Sn/amorphous-carbon (Sn/a-C), and SnBi/amorphous carbon (SnBi/a-C) microparticles (MPs) were successfully synthesized. The thermal stabilities of Sn/a-C and SnBi/a-C core–shell MPs were verified by cycling tests, and stable latent heats of 56 and 45.7 J/g were obtained for Sn/a-C and SnBi/a-C MPs, respectively. Compared to the high melting point of 231 °C and large thermal hysteresis (TH) of ∼106 °C for the Sn/a-C MPs, the SnBi/a-C MPs exhibited a lower melting point of 125 °C and a smaller TH of 20 °C. The nitrate senary salt with a lower melting point of ∼75 °C than that of the commercial HITEC salt (melting point of ∼142 °C) was also synthesized to enlarge the working temperature range of the working fluid in a solar thermal power plant and to demonstrate the latent heat-enhanced thermal energy storage using the SnBi/a-C MPs. The heat capacity can be enhanced by 200% by doping with 20 wt % Sn/a-C MPs into the HITEC salt, and it can be enhanced by 734% by doping with 20 wt % SnBi/a-C MPs into the senary salt. In addition, the viscosities of the HITEC salt and senary salt doped with the Sn/a-C and SnBi/a-C MPs were not appreciably raised by doping with the MPs. The various approaches accomplished in this work demonstrate (1) enhancing heat capacity of the working fluid by exploiting the latent heats of the embedded MPs; (2) lowering the TH of the MPs by using the alloy metal particles; and (3) extending the working temperature range by synthesizing the senary salt. These approaches could be applied for enhancing energy storage in solar thermal power plants and facilitating waste heat recovery., Peer reviewed

Proyecto: //
DOI: http://hdl.handle.net/10261/331918
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331918
HANDLE: http://hdl.handle.net/10261/331918
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331918
PMID: http://hdl.handle.net/10261/331918
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331918
Ver en: http://hdl.handle.net/10261/331918
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331918

Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331919
Dataset. 2022

COBALT MOLYBDENUM NITRIDE-BASED NANOSHEETS FOR SEAWATER SPLITTING [DATASET]

  • Wang, Xiang
  • Han, Xu
  • Du, Ruifeng
  • Xing, Congcong
  • Qi, Xueqiang
  • Liang, Zhifu
  • Guardia, Pablo
  • Arbiol, Jordi
  • Cabot, Andreu
  • Li, Junshan
22 pages. -- PDF file includes: Structural characterization. -- Electrochemical Measurements. -- Faradaic efficiency (FE). -- Ionic strength compensation. -- Hypochlorite titration analysis. -- DFT Computational method. -- Figure S1 a-f) SEM images of the Mo-Co precursor. -- Figure S2 a-f) SEM images of MoN-Co2N. -- Figure S3. a-c) SEM images and d) EDS result of MoN-Co2N. -- Figure S4. a-f) TEM images of MoN-Co2N. -- Figure S5. a-c) TEM images and d-f) HAADF-STEM images of MoN-Co2N. -- Figure S6. TEM and HRTEM images of the MoN-Co2N. -- Figure S7. EELS chemical composition maps of MoN-Co2N obtained from the red squared area of the STEM micrograph. -- Figure S8. XRD patterns of MoN-Co2N, Mo-Co2N and CoxN compared with reference patterns of Co, Co4N, Co2N and MoN. -- Figure S9. a-c) SEM images and d) EDS result of Mo-Co2N. -- Figure S10. a-c) SEM images and d) EDS result of CoxN. -- Figure S11. Pore size distribution plot for MoN-Co2N and CoxN. -- Figure S12. Cyclic voltammograms for a) MoN-Co2N; b) Mo-Co2N; c) CoxN and d) Mo-Co precursors and e) IrO2 in the non-faradaic region of 1.12-1.22 V vs. RHE at various scan rates. -- Figure 13. a) N and b) Co partial and c) Total Density of states for Co2N and MoN-Co2N. -- Figure S14. The evolutions of local structural configurations for illustrating OER process of Co2N. (* represents the active site). -- Figure S15. Photograph of flasks containing seawater after adding KOH (left) and before adding KOH (right). -- Figure S16. Overall water/seawater splitting performance of MoN-Co2N ‖ MoN-Co2N and Pt/C ‖ IrO2 cells in 1 M KOH, 1 M KOH seawater, and untreated seawater. -- Figure S17. Overall water splitting performance of MoN-Co2N ‖ MoN-Co2N and Pt/C ‖ IrO2 cells in 1 M KOH seawater with and without ionic strength compensation. -- Figure S18. a) Digital photographs of the reaction and reference solutions for the iodometric titration, showing the absence of ClO− production in theº former case. b) Measured (dots) and theoretical (solid line) gaseous products by the two-electrode electrolyzer at a current of 50 mA cm-2 in 1 M KOH seawater. -- Figure S19. High-resolution XPS spectra of a) N 1s, b) Co 2p, c) Mo 3d for MoN-Co2N and MoN-Co2N after long-term reaction. -- Figure S20. a-b) TEM images, c-d) HAADF-STEM images, e-f) HRTEM of MoN-Co2N after long time reaction. -- Figure S21. EELS chemical composition maps of MoN-Co2N after long time reaction obtained from the red squared area of the STEM micrograph. Individual C K-edge at 284 eV (orange), N K-edge at 401 eV (blue), Co L2,3-edges at 779 eV (red) and Mo M4,5-edges at 227 eV (green) and composites of Co-N and Mo-N. -- Table S1. Comparison of OER activity of MoN-Co2N with recently reported nitride electrocatalysts in 1.0 M KOH electrolyte. -- Table S2. Cdl and ECSAs of various catalysts. -- Table S3. Comparison of HER performance of MoN-Co2N with recently reported nitride electrocatalysts in 1.0 M KOH electrolyte. -- Table S4 Comparison of the overall water splitting of MoN-Co2N with previously reported electrocatalysts., The development of cost-effective bifunctional catalysts for water electrolysis is both a crucial necessity and an exciting scientific challenge. Herein, a simple approach based on a metal–organic framework sacrificial template to preparing cobalt molybdenum nitride supported on nitrogen-doped carbon nanosheets is reported. The porous structure of produced composite enables fast reaction kinetics, enhanced stability, and high corrosion resistance in critical seawater conditions. The cobalt molybdenum nitride-based electrocatalyst is tested toward both oxygen evolution reaction and hydrogen evolution reaction half-reactions using the seawater electrolyte, providing excellent performances that are rationalized using density functional theory. Subsequently, the nitride composite is tested as a bifunctional catalyst for the overall splitting of KOH-treated seawater from the Mediterranean Sea. The assembled system requires overpotentials of just 1.70 V to achieve a current density of 100 mA cm–2 in 1 M KOH seawater and continuously works for over 62 h. This work demonstrates the potential of transition-metal nitrides for seawater splitting and represents a step forward toward the cost-effective implementation of this technology., Peer reviewed

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DOI: http://hdl.handle.net/10261/331919
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331919
HANDLE: http://hdl.handle.net/10261/331919
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331919
PMID: http://hdl.handle.net/10261/331919
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331919
Ver en: http://hdl.handle.net/10261/331919
Digital.CSIC. Repositorio Institucional del CSIC
oai:digital.csic.es:10261/331919

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